Telomere length: From cellular senescence to trajectories of human aging (notice n° 173327)
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| 000 -LEADER | |
|---|---|
| fixed length control field | 01636cam a2200253 4500500 |
| 005 - DATE AND TIME OF LATEST TRANSACTION | |
| control field | 20250112035638.0 |
| 041 ## - LANGUAGE CODE | |
| Language code of text/sound track or separate title | fre |
| 042 ## - AUTHENTICATION CODE | |
| Authentication code | dc |
| 100 10 - MAIN ENTRY--PERSONAL NAME | |
| Personal name | Benetos, Athanase |
| Relator term | author |
| 245 00 - TITLE STATEMENT | |
| Title | Telomere length: From cellular senescence to trajectories of human aging |
| 260 ## - PUBLICATION, DISTRIBUTION, ETC. | |
| Date of publication, distribution, etc. | 2022.<br/> |
| 500 ## - GENERAL NOTE | |
| General note | 64 |
| 520 ## - SUMMARY, ETC. | |
| Summary, etc. | The last two decades have made it possible to understand the role of telomeres in the aging process and in longevity via their direct influence on replicative senescence and the capacity for tissue repair. Telomere length is primarily determined by genetic factors and secondarily by the influence of environmental factors during the first years of life. Short telomeres have a causal role in the development of age-related degenerative diseases and reduced longevity. Therefore, telomere length can be considered as a “biological capital” formed early in life, which plays an important role in the damage/repair balance at the cellular and tissue level and can thus influence the trajectory of aging. The integration of telomere length into a set of clinical, biological, and social data would make it possible to better individualize strategies for the prevention of age-related diseases, frailty, and loss of autonomy. |
| 690 ## - LOCAL SUBJECT ADDED ENTRY--TOPICAL TERM (OCLC, RLIN) | |
| Topical term or geographic name as entry element | cellular senescence |
| 690 ## - LOCAL SUBJECT ADDED ENTRY--TOPICAL TERM (OCLC, RLIN) | |
| Topical term or geographic name as entry element | age-related diseases |
| 690 ## - LOCAL SUBJECT ADDED ENTRY--TOPICAL TERM (OCLC, RLIN) | |
| Topical term or geographic name as entry element | telomeres |
| 690 ## - LOCAL SUBJECT ADDED ENTRY--TOPICAL TERM (OCLC, RLIN) | |
| Topical term or geographic name as entry element | aging |
| 690 ## - LOCAL SUBJECT ADDED ENTRY--TOPICAL TERM (OCLC, RLIN) | |
| Topical term or geographic name as entry element | Ageing |
| 690 ## - LOCAL SUBJECT ADDED ENTRY--TOPICAL TERM (OCLC, RLIN) | |
| Topical term or geographic name as entry element | Telomeres |
| 690 ## - LOCAL SUBJECT ADDED ENTRY--TOPICAL TERM (OCLC, RLIN) | |
| Topical term or geographic name as entry element | Age-related diseases |
| 690 ## - LOCAL SUBJECT ADDED ENTRY--TOPICAL TERM (OCLC, RLIN) | |
| Topical term or geographic name as entry element | Cellular senescence |
| 786 0# - DATA SOURCE ENTRY | |
| Note | Hegel | - | 3 | 2022-11-08 | p. 269-280 | 2269-0530 |
| 856 41 - ELECTRONIC LOCATION AND ACCESS | |
| Uniform Resource Identifier | <a href="https://shs.cairn.info/journal-hegel-2022-3-page-269?lang=en">https://shs.cairn.info/journal-hegel-2022-3-page-269?lang=en</a> |
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