Carence martiale sans anémie : première comorbidité curable de l’insuffisance cardiaque (notice n° 275697)
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| fixed length control field | 04732cam a2200361 4500500 |
| 005 - DATE AND TIME OF LATEST TRANSACTION | |
| control field | 20250117193439.0 |
| 041 ## - LANGUAGE CODE | |
| Language code of text/sound track or separate title | fre |
| 042 ## - AUTHENTICATION CODE | |
| Authentication code | dc |
| 100 10 - MAIN ENTRY--PERSONAL NAME | |
| Personal name | Peoc’h, Katell |
| Relator term | author |
| 245 00 - TITLE STATEMENT | |
| Title | Carence martiale sans anémie : première comorbidité curable de l’insuffisance cardiaque |
| 260 ## - PUBLICATION, DISTRIBUTION, ETC. | |
| Date of publication, distribution, etc. | 2022.<br/> |
| 500 ## - GENERAL NOTE | |
| General note | 93 |
| 520 ## - SUMMARY, ETC. | |
| Summary, etc. | Le fonctionnement du muscle cardiaque est particulièrement sensible à la carence martiale, comorbidité aisément curable la plus fréquemment associée à l’insuffisance cardiaque. Les patients insuffisants cardiaques carencés en fer sont plus souvent ré-hospitalisés et ont une espérance de vie réduite. Des études randomisées récentes ont montré que l’apport de fer exogène s’accompagnait d’une amélioration de la capacité fonctionnelle (test de marche), de la qualité de vie et du taux de ré-hospitalisation de ces patients. Les symptômes de la carence martiale sont peu spécifiques et souvent confondus avec ceux de l’insuffisance cardiaque ou des autres comorbidités, ce qui explique la prise en charge souvent trop tardive de ce déficit. L’anémie n’est qu’une conséquence tardive de cette carence martiale. Du fait de l’état inflammatoire associé à l’insuffisance cardiaque chronique, seule la voie parentérale permet de court-circuiter la séquestration tissulaire macrophagique du fer et l’inhibition de son absorption intestinale. Les récentes recommandations européennes préconisent un dépistage de la carence martiale (ferritine sérique et coefficient de saturation de la transferrine) chez tous les patients suspectés d’insuffisance cardiaque, un bilan martial systématique chez tous les patients insuffisants cardiaques, ainsi qu’une supplémentation en fer par voie intraveineuse en cas de carence chez les patients symptomatiques. Étant donné le caractère péjoratif de la carence martiale sur l’évolution de la maladie, la fréquence et l’impact financier des hospitalisations liées aux épisodes de décompensation ainsi que l’efficacité d’une simple supplémentation, le dépistage de cette comorbidité doit désormais faire partie des examens de routine chez tous les patients insuffisants cardiaques. |
| 520 ## - SUMMARY, ETC. | |
| Summary, etc. | The functioning of the heart muscle is particularly sensitive to iron deficiency, the easily curable comorbidity most frequently associated with heart failure. Iron-deficient heart failure patients are more often rehospitalized and have reduced survival. Heart muscle function is particularly susceptible to martial deficiency. Recent randomized studies have shown that exogenous iron intake is accompanied by improved functional capacity (walking test), quality of life, and re-hospitalization rate in these patients. The symptoms of iron deficiency are not very specific and often confused with those of heart failure or other comorbidities, which explains why management is often too late. Anemia is only a late consequence of this iron deficiency. Due to the inflammatory state associated with chronic heart failure, only the parenteral route can bypass the macrophage tissue sequestration of iron and inhibit its intestinal absorption. Recent European guidelines recommend screening for iron deficiency (serum ferritin and transferrin saturation coefficient) in all patients with suspected heart failure, routine iron parameters assessment in all patients with heart failure, and intravenous iron supplementation in case of deficiency in symptomatic patients. Given the pejorative nature of iron deficiency on disease progression, the frequency and financial impact of hospitalizations linked to episodes of decompensation, as well as the effectiveness of simple supplementation, screening for this comorbidity, screening for this frequent comorbidity should now be part of routine testing in all heart failure patients. |
| 690 ## - LOCAL SUBJECT ADDED ENTRY--TOPICAL TERM (OCLC, RLIN) | |
| Topical term or geographic name as entry element | carboxymaltose ferrique |
| 690 ## - LOCAL SUBJECT ADDED ENTRY--TOPICAL TERM (OCLC, RLIN) | |
| Topical term or geographic name as entry element | carence martiale |
| 690 ## - LOCAL SUBJECT ADDED ENTRY--TOPICAL TERM (OCLC, RLIN) | |
| Topical term or geographic name as entry element | ferritine sérique |
| 690 ## - LOCAL SUBJECT ADDED ENTRY--TOPICAL TERM (OCLC, RLIN) | |
| Topical term or geographic name as entry element | insuffisance cardiaque |
| 690 ## - LOCAL SUBJECT ADDED ENTRY--TOPICAL TERM (OCLC, RLIN) | |
| Topical term or geographic name as entry element | coefficient de saturation de la transferrine |
| 690 ## - LOCAL SUBJECT ADDED ENTRY--TOPICAL TERM (OCLC, RLIN) | |
| Topical term or geographic name as entry element | serum ferritin |
| 690 ## - LOCAL SUBJECT ADDED ENTRY--TOPICAL TERM (OCLC, RLIN) | |
| Topical term or geographic name as entry element | transferrin saturation coefficient |
| 690 ## - LOCAL SUBJECT ADDED ENTRY--TOPICAL TERM (OCLC, RLIN) | |
| Topical term or geographic name as entry element | martial deficiency |
| 690 ## - LOCAL SUBJECT ADDED ENTRY--TOPICAL TERM (OCLC, RLIN) | |
| Topical term or geographic name as entry element | ferric carboxymaltose |
| 690 ## - LOCAL SUBJECT ADDED ENTRY--TOPICAL TERM (OCLC, RLIN) | |
| Topical term or geographic name as entry element | heart failure |
| 700 10 - ADDED ENTRY--PERSONAL NAME | |
| Personal name | Ausseil, Jérôme |
| Relator term | author |
| 700 10 - ADDED ENTRY--PERSONAL NAME | |
| Personal name | Feugeas, Jean-Paul |
| Relator term | author |
| 700 10 - ADDED ENTRY--PERSONAL NAME | |
| Personal name | Guieu, Régis |
| Relator term | author |
| 700 10 - ADDED ENTRY--PERSONAL NAME | |
| Personal name | Masson, Damien |
| Relator term | author |
| 700 10 - ADDED ENTRY--PERSONAL NAME | |
| Personal name | Sablonnière, Bernard |
| Relator term | author |
| 700 10 - ADDED ENTRY--PERSONAL NAME | |
| Personal name | Puy, Hervé |
| Relator term | author |
| 786 0# - DATA SOURCE ENTRY | |
| Note | Annales de Biologie Clinique | 80 | 2 | 2022-03-01 | p. 109-118 | 0003-3898 |
| 856 41 - ELECTRONIC LOCATION AND ACCESS | |
| Uniform Resource Identifier | <a href="https://shs.cairn.info/revue-annales-de-biologie-clinique-2022-2-page-109?lang=fr&redirect-ssocas=7080">https://shs.cairn.info/revue-annales-de-biologie-clinique-2022-2-page-109?lang=fr&redirect-ssocas=7080</a> |
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