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Today, according to the Analgesia Institute, France's leading research foundation for pain-management research and innovation, one in five adults suffers from chronic pain and two out of three patients do not receive any relief from their treatment.However, as early as 1917, Sherrington offered an essential clue to understanding chronic pain, when he debunked the notion that reflex reactions are designed to restore homeostasis. According to Sherrington’s law of reciprocal innervation, “when agonists contract, antagonists relax." We now know that the only way to restore the proper functioning of a pair of forces is to inhibit the agonist by means of contraction, so that it in turn inhibits the antagonist.Among the thousands of publications on the trigger point available on PubMed, it seems that one aspect has not been taken into account: the inclusion of the biological tensegrity model.Through this approach, I propose to consider another way of treating chronic pain: artificially producing a withdrawal reflex on the tendon of the agonist muscle responsible for the autogenic inhibition reflex to reprogram the force couple.This would therefore allow tenotomy, analgesia, neurostimulation, and neurotomy to be avoided, and for definitive results on chronic pain based on the all-or-none law to be obtained. If my theory of reflex post-traumatic amnesia proves to be correct, it would restart an organic function in default mode. Thus, it would be a curative innovation for any pathology of functional and/or organic origin.