000 | 01367cam a2200325 4500500 | ||
---|---|---|---|
005 | 20250112065611.0 | ||
041 | _afre | ||
042 | _adc | ||
100 | 1 | 0 |
_aLombardo, Giulia _eauthor |
700 | 1 | 0 |
_a Marabini, Laura _eauthor |
700 | 1 | 0 |
_a Doneda, Luisa _eauthor |
700 | 1 | 0 |
_a Lombardo, Vincenza _eauthor |
700 | 1 | 0 |
_a Scricciolo, Alice _eauthor |
700 | 1 | 0 |
_a Elli, Luca _eauthor |
700 | 1 | 0 |
_a Della Valle, Valentina _eauthor |
700 | 1 | 0 |
_a Muratori, Simona _eauthor |
700 | 1 | 0 |
_a Roncoroni, Leda _eauthor |
245 | 0 | 0 | _aDNA damage in human skin fibroblasts from patients with dermatitis herpetiformis |
260 | _c2019. | ||
500 | _a76 | ||
520 | _aBackgroundDermatitis herpetiformis (DH) and celiac disease (CD) are considered to be autoimmune diseases that share a specific trigger (gluten) and a common genetic background (HLA-DQ2/DQ8). However, the pathogenesis of DH is not yet fully understood and no data are available regarding a possible role of fibroblasts in this disease. | ||
690 | _a33mer peptide | ||
690 | _agenotoxicity | ||
690 | _adapsone | ||
690 | _aceliac disease | ||
690 | _aprimary fibroblasts | ||
690 | _agliadin | ||
786 | 0 | _nEuropean Journal of Dermatology | 29 | 2 | 2019-03-01 | p. 167-173 | 1167-1122 | |
856 | 4 | 1 | _uhttps://shs.cairn.info/revue-european-journal-of-dermatology-2019-2-page-167?lang=en |
999 |
_c242538 _d242538 |