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041 _afre
042 _adc
100 1 0 _aLombardo, Giulia
_eauthor
700 1 0 _a Marabini, Laura
_eauthor
700 1 0 _a Doneda, Luisa
_eauthor
700 1 0 _a Lombardo, Vincenza
_eauthor
700 1 0 _a Scricciolo, Alice
_eauthor
700 1 0 _a Elli, Luca
_eauthor
700 1 0 _a Della Valle, Valentina
_eauthor
700 1 0 _a Muratori, Simona
_eauthor
700 1 0 _a Roncoroni, Leda
_eauthor
245 0 0 _aDNA damage in human skin fibroblasts from patients with dermatitis herpetiformis
260 _c2019.
500 _a76
520 _aBackgroundDermatitis herpetiformis (DH) and celiac disease (CD) are considered to be autoimmune diseases that share a specific trigger (gluten) and a common genetic background (HLA-DQ2/DQ8). However, the pathogenesis of DH is not yet fully understood and no data are available regarding a possible role of fibroblasts in this disease.
690 _a33mer peptide
690 _agenotoxicity
690 _adapsone
690 _aceliac disease
690 _aprimary fibroblasts
690 _agliadin
786 0 _nEuropean Journal of Dermatology | 29 | 2 | 2019-03-01 | p. 167-173 | 1167-1122
856 4 1 _uhttps://shs.cairn.info/revue-european-journal-of-dermatology-2019-2-page-167?lang=en
999 _c242538
_d242538